Sodium accumulation during ischemia induces mitochondrial damage in perfused rat hearts

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Sodium accumulation during ischemia induces mitochondrial damage in perfused rat hearts.

OBJECTIVE The present study aimed to elucidate the involvement of sodium overload and following damage to mitochondria during ischemia in the genesis of ischemia/reperfusion injury of perfused rat hearts. METHODS Isolated, perfused hearts were exposed to different durations (15-35 min) of ischemia followed by 60-min reperfusion. At the end of ischemia or reperfusion, myocardial sodium and cal...

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Real-time 2-photon imaging of mitochondrial function in perfused rat hearts subjected to ischemia/reperfusion.

BACKGROUND Mitochondria play pivotal roles in cell death; the loss of mitochondrial membrane potential (delta psi(m)) is the earliest event that commits the cell to death. Here, we report novel real-time imaging of delta psi(m) in individual cardiomyocytes within perfused rat hearts using 2-photon laser-scanning microscopy, which has unique advantages over conventional confocal microscopy: grea...

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Classic preconditioning decreases the harmful accumulation of nitric oxide during ischemia and reperfusion in rat hearts.

BACKGROUND The role of NO in the mechanism of preconditioning is not understood. Therefore, we studied the effect of preconditioning and subsequent ischemia/reperfusion on myocardial NO content in the presence of an NO synthase (NOS) inhibitor. METHODS AND RESULTS Isolated working rat hearts were subjected to preconditioning protocols of 3 intermittent periods of rapid pacing or no-flow ische...

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ژورنال

عنوان ژورنال: Cardiovascular Research

سال: 2002

ISSN: 0008-6363

DOI: 10.1016/s0008-6363(02)00282-1